Ras oncogene-independent activation of RALB signaling is a targetable mechanism of escape from NRAS(V12) oncogene addiction in acute myeloid leukemia. [electronic resource]
Producer: 20171011Description: 3263-3273 p. digitalISSN:- 1476-5594
- Animals
- Apoptosis
- Biomarkers, Tumor -- genetics
- Cell Proliferation
- Female
- GTP Phosphohydrolases -- genetics
- Gene Expression Regulation, Neoplastic
- Humans
- Leukemia, Experimental -- genetics
- Leukemia, Myeloid, Acute -- genetics
- Male
- Membrane Proteins -- genetics
- Mice
- Mice, SCID
- Mitogen-Activated Protein Kinases -- genetics
- Monomeric GTP-Binding Proteins -- genetics
- Mutation -- genetics
- Neoplasm Invasiveness
- Oncogenes
- Signal Transduction
- Tumor Cells, Cultured
- Xenograft Model Antitumor Assays
- ral GTP-Binding Proteins -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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