AP-1 regulates cyclin D1 and c-MYC transcription in an AKT-dependent manner in response to mTOR inhibition: role of AIP4/Itch-mediated JUNB degradation. [electronic resource]
Producer: 20110506Description: 115-30 p. digitalISSN:- 1557-3125
- Animals
- Antibiotics, Antineoplastic -- pharmacology
- Cell Line
- Cell Line, Tumor
- Cells, Cultured
- Cyclin D1 -- genetics
- Gene Expression -- drug effects
- Humans
- Immunoblotting
- Mice
- Mice, Knockout
- Models, Genetic
- PTEN Phosphohydrolase -- genetics
- Promoter Regions, Genetic -- genetics
- Protein Binding -- drug effects
- Proto-Oncogene Proteins c-akt -- genetics
- Proto-Oncogene Proteins c-jun -- genetics
- Proto-Oncogene Proteins c-myc -- genetics
- RNA Interference
- Repressor Proteins -- genetics
- Reverse Transcriptase Polymerase Chain Reaction
- Sirolimus -- pharmacology
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
- Transcription Factor AP-1 -- genetics
- Ubiquitin-Protein Ligases -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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