MSH2 in contrast to MLH1 and MSH6 is frequently inactivated by exonic and promoter rearrangements in hereditary nonpolyposis colorectal cancer. [electronic resource]
Producer: 20020305Description: 848-53 p. digitalISSN:- 0008-5472
- Adaptor Proteins, Signal Transducing
- Carrier Proteins
- Colorectal Neoplasms, Hereditary Nonpolyposis -- genetics
- DNA-Binding Proteins -- genetics
- Exons
- Gene Expression Regulation, Neoplastic
- Gene Rearrangement
- Gene Silencing
- Humans
- Middle Aged
- MutL Protein Homolog 1
- MutS Homolog 2 Protein
- Neoplasm Proteins -- genetics
- Nuclear Proteins
- Point Mutation
- Polymerase Chain Reaction -- methods
- Promoter Regions, Genetic -- genetics
- Proto-Oncogene Proteins -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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