Potential triple helix-mediated inhibition of IGF-I gene expression significantly reduces tumorigenicity of glioblastoma in an animal model. [electronic resource]
Producer: 19970612Description: 105-12 p. digitalISSN:- 0929-1903
- Amyloid beta-Protein Precursor
- Animals
- Carrier Proteins -- drug effects
- Disease Models, Animal
- Gene Expression Regulation, Neoplastic -- drug effects
- Genetic Therapy -- methods
- Glioblastoma -- drug therapy
- Histocompatibility Antigens Class I -- drug effects
- Insulin-Like Growth Factor I -- chemistry
- Mice
- Mice, Nude
- Neoplasms, Experimental -- genetics
- Nucleic Acid Conformation
- Oligonucleotides -- chemistry
- Plasmids -- chemistry
- Protease Nexins
- Protein Biosynthesis
- Rats
- Receptors, Cell Surface
- Transfection
- Tumor Cells, Cultured
- Up-Regulation
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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