Potential triple helix-mediated inhibition of IGF-I gene expression significantly reduces tumorigenicity of glioblastoma in an animal model.
Shevelev, A
Potential triple helix-mediated inhibition of IGF-I gene expression significantly reduces tumorigenicity of glioblastoma in an animal model. [electronic resource] - Cancer gene therapy - 105-12 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0929-1903
Amyloid beta-Protein Precursor
Animals
Carrier Proteins--drug effects
Disease Models, Animal
Gene Expression Regulation, Neoplastic--drug effects
Genetic Therapy--methods
Glioblastoma--drug therapy
Histocompatibility Antigens Class I--drug effects
Insulin-Like Growth Factor I--chemistry
Mice
Mice, Nude
Neoplasms, Experimental--genetics
Nucleic Acid Conformation
Oligonucleotides--chemistry
Plasmids--chemistry
Protease Nexins
Protein Biosynthesis
Rats
Receptors, Cell Surface
Transfection
Tumor Cells, Cultured
Up-Regulation
Potential triple helix-mediated inhibition of IGF-I gene expression significantly reduces tumorigenicity of glioblastoma in an animal model. [electronic resource] - Cancer gene therapy - 105-12 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0929-1903
Amyloid beta-Protein Precursor
Animals
Carrier Proteins--drug effects
Disease Models, Animal
Gene Expression Regulation, Neoplastic--drug effects
Genetic Therapy--methods
Glioblastoma--drug therapy
Histocompatibility Antigens Class I--drug effects
Insulin-Like Growth Factor I--chemistry
Mice
Mice, Nude
Neoplasms, Experimental--genetics
Nucleic Acid Conformation
Oligonucleotides--chemistry
Plasmids--chemistry
Protease Nexins
Protein Biosynthesis
Rats
Receptors, Cell Surface
Transfection
Tumor Cells, Cultured
Up-Regulation