Vasoactive intestinal peptide (VIP) and pituitary adenylate cyclase-activating polypeptide (PACAP) potentiate the glutamate-evoked release of arachidonic acid from mouse cortical neurons. Evidence for a cAMP-independent mechanism. [electronic resource]
Producer: 19961118Description: 23705-10 p. digitalISSN:- 0021-9258
- Animals
- Arachidonic Acid -- metabolism
- Calcium -- physiology
- Cells, Cultured
- Cerebral Cortex -- metabolism
- Cyclic AMP -- physiology
- Cyclic AMP-Dependent Protein Kinases -- physiology
- GTP-Binding Protein alpha Subunits, Gs -- physiology
- Glutamates -- physiology
- Mice
- Neuropeptides -- physiology
- Neurotransmitter Agents -- physiology
- Pituitary Adenylate Cyclase-Activating Polypeptide
- Receptors, AMPA -- physiology
- Receptors, N-Methyl-D-Aspartate -- physiology
- Receptors, Pituitary Adenylate Cyclase-Activating Polypeptide
- Receptors, Pituitary Hormone -- physiology
- Signal Transduction
- Type C Phospholipases -- physiology
- Vasoactive Intestinal Peptide -- physiology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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