Vasoactive intestinal peptide (VIP) and pituitary adenylate cyclase-activating polypeptide (PACAP) potentiate the glutamate-evoked release of arachidonic acid from mouse cortical neurons. Evidence for a cAMP-independent mechanism.

Stella, N

Vasoactive intestinal peptide (VIP) and pituitary adenylate cyclase-activating polypeptide (PACAP) potentiate the glutamate-evoked release of arachidonic acid from mouse cortical neurons. Evidence for a cAMP-independent mechanism. [electronic resource] - The Journal of biological chemistry Sep 1996 - 23705-10 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0021-9258

10.1074/jbc.271.39.23705 doi


Animals
Arachidonic Acid--metabolism
Calcium--physiology
Cells, Cultured
Cerebral Cortex--metabolism
Cyclic AMP--physiology
Cyclic AMP-Dependent Protein Kinases--physiology
GTP-Binding Protein alpha Subunits, Gs--physiology
Glutamates--physiology
Mice
Neuropeptides--physiology
Neurotransmitter Agents--physiology
Pituitary Adenylate Cyclase-Activating Polypeptide
Receptors, AMPA--physiology
Receptors, N-Methyl-D-Aspartate--physiology
Receptors, Pituitary Adenylate Cyclase-Activating Polypeptide
Receptors, Pituitary Hormone--physiology
Signal Transduction
Type C Phospholipases--physiology
Vasoactive Intestinal Peptide--physiology