The multiple endocrine neoplasia type 2B point mutation alters long-term regulation and enhances the transforming capacity of the epidermal growth factor receptor. [electronic resource]
Producer: 19960620Description: 5850-8 p. digitalISSN:- 0021-9258
- 3T3 Cells
- Amino Acid Sequence
- Animals
- Base Sequence
- Cell Division -- drug effects
- Cell Line
- Cell Transformation, Neoplastic
- DNA Primers
- Down-Regulation
- Drosophila Proteins
- Epidermal Growth Factor -- metabolism
- ErbB Receptors -- biosynthesis
- Gene Expression Regulation, Neoplastic
- Gene Library
- Humans
- Kinetics
- Mice
- Molecular Sequence Data
- Multiple Endocrine Neoplasia Type 2b -- genetics
- Mutagenesis, Site-Directed
- Peptides -- chemical synthesis
- Phosphorylation
- Plasmids
- Point Mutation
- Proto-Oncogene Mas
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins c-ret
- Proto-Oncogenes
- Receptor Protein-Tyrosine Kinases -- genetics
- Recombinant Proteins -- biosynthesis
- Substrate Specificity
- Tumor Cells, Cultured
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Publication Type: Comparative Study; Journal Article
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