Loss of Na+ channel inactivation by anemone toxin (ATX II) mimics the myotonic state in hyperkalaemic periodic paralysis. [electronic resource]
Producer: 19931122Description: 501-20 p. digitalISSN:- 0022-3751
- Action Potentials -- drug effects
- Animals
- Cnidarian Venoms -- pharmacology
- Disease Models, Animal
- Female
- Humans
- Hyperkalemia -- etiology
- In Vitro Techniques
- Isometric Contraction -- drug effects
- Muscles -- drug effects
- Mutation
- Myotonia -- etiology
- Paralyses, Familial Periodic -- etiology
- Rats
- Sea Anemones
- Sodium Channel Blockers
- Sodium Channels -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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