Loss of Na+ channel inactivation by anemone toxin (ATX II) mimics the myotonic state in hyperkalaemic periodic paralysis.

Cannon, S C

Loss of Na+ channel inactivation by anemone toxin (ATX II) mimics the myotonic state in hyperkalaemic periodic paralysis. [electronic resource] - The Journal of physiology Jul 1993 - 501-20 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0022-3751


Action Potentials--drug effects
Animals
Cnidarian Venoms--pharmacology
Disease Models, Animal
Female
Humans
Hyperkalemia--etiology
In Vitro Techniques
Isometric Contraction--drug effects
Muscles--drug effects
Mutation
Myotonia--etiology
Paralyses, Familial Periodic--etiology
Rats
Sea Anemones
Sodium Channel Blockers
Sodium Channels--genetics