Thrombin activation of PAR-1 contributes to microvascular stasis in mouse models of sickle cell disease. [electronic resource]
Producer: 20201222Description: 1783-1787 p. digitalISSN:- 1528-0020
- Anemia, Sickle Cell -- complications
- Animals
- Blood Coagulation Disorders -- etiology
- Blood Platelets -- metabolism
- Constriction, Pathologic -- genetics
- Disease Models, Animal
- Female
- Hemoglobin, Sickle -- genetics
- Humans
- Male
- Mice
- Mice, Transgenic
- Microvessels -- metabolism
- Receptor, PAR-1 -- genetics
- Thrombin -- metabolism
- Vascular Diseases -- etiology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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