Thrombin activation of PAR-1 contributes to microvascular stasis in mouse models of sickle cell disease.
Sparkenbaugh, Erica M
Thrombin activation of PAR-1 contributes to microvascular stasis in mouse models of sickle cell disease. [electronic resource] - Blood 05 2020 - 1783-1787 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1528-0020
10.1182/blood.2019003543 doi
Anemia, Sickle Cell--complications
Animals
Blood Coagulation Disorders--etiology
Blood Platelets--metabolism
Constriction, Pathologic--genetics
Disease Models, Animal
Female
Hemoglobin, Sickle--genetics
Humans
Male
Mice
Mice, Transgenic
Microvessels--metabolism
Receptor, PAR-1--genetics
Thrombin--metabolism
Vascular Diseases--etiology
Thrombin activation of PAR-1 contributes to microvascular stasis in mouse models of sickle cell disease. [electronic resource] - Blood 05 2020 - 1783-1787 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1528-0020
10.1182/blood.2019003543 doi
Anemia, Sickle Cell--complications
Animals
Blood Coagulation Disorders--etiology
Blood Platelets--metabolism
Constriction, Pathologic--genetics
Disease Models, Animal
Female
Hemoglobin, Sickle--genetics
Humans
Male
Mice
Mice, Transgenic
Microvessels--metabolism
Receptor, PAR-1--genetics
Thrombin--metabolism
Vascular Diseases--etiology