A study on the mechanism of rapamycin mediating the sensitivity of pancreatic cancer cells to cisplatin through PI3K/AKT/mTOR signaling pathway. [electronic resource]
Producer: 20191121Description: 739-745 p. digitalISSN:- 2241-6293
- Animals
- Antineoplastic Combined Chemotherapy Protocols -- pharmacology
- Apoptosis -- drug effects
- Autophagy -- genetics
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Cisplatin -- pharmacology
- Drug Resistance, Neoplasm -- drug effects
- Gene Expression Regulation, Neoplastic
- Humans
- Pancreatic Neoplasms -- drug therapy
- Phosphatidylinositol 3-Kinases -- genetics
- Phosphorylation -- drug effects
- Proto-Oncogene Proteins c-akt -- genetics
- Signal Transduction -- drug effects
- Sirolimus -- pharmacology
- TOR Serine-Threonine Kinases -- genetics
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Publication Type: Journal Article
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