Complement C3aR Inactivation Attenuates Tau Pathology and Reverses an Immune Network Deregulated in Tauopathy Models and Alzheimer's Disease. [electronic resource]
Producer: 20190808Description: 1337-1353.e5 p. digitalISSN:- 1097-4199
- Adult
- Aged
- Aged, 80 and over
- Alzheimer Disease -- complications
- Animals
- Brain -- pathology
- Calcium-Binding Proteins
- Cognition Disorders -- etiology
- Complement C3a -- genetics
- Cytokines -- metabolism
- DNA-Binding Proteins -- metabolism
- Disease Models, Animal
- Evoked Potentials -- physiology
- Female
- Gene Regulatory Networks -- physiology
- Humans
- Male
- Mice
- Mice, Transgenic
- Microfilament Proteins
- Middle Aged
- Receptors, Complement -- genetics
- Signal Transduction -- drug effects
- Tauopathies -- complications
- Up-Regulation -- physiology
- tau Proteins -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.