Impairment of Nitric Oxide Pathway by Intravascular Hemolysis Plays a Major Role in Mice Esophageal Hypercontractility: Reversion by Soluble Guanylyl Cyclase Stimulator. [electronic resource]
Producer: 20191008Description: 194-202 p. digitalISSN:- 1521-0103
- Anemia, Sickle Cell -- drug therapy
- Animals
- Cyclic GMP -- metabolism
- Esophageal Diseases -- drug therapy
- Esophagus -- drug effects
- Guanylate Cyclase -- metabolism
- Hemolysis -- drug effects
- Male
- Mice
- Mice, Inbred C57BL
- Models, Animal
- Muscle Contraction -- drug effects
- Muscle, Smooth -- drug effects
- Nitric Oxide -- metabolism
- Nitric Oxide Donors -- pharmacology
- Nitric Oxide Synthase Type III -- metabolism
- Nitroprusside -- pharmacology
- Oxidative Stress -- drug effects
- Phenylhydrazines -- pharmacology
- Pyrazoles -- pharmacology
- Pyridines -- metabolism
- Signal Transduction -- drug effects
- Soluble Guanylyl Cyclase -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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