Impairment of Nitric Oxide Pathway by Intravascular Hemolysis Plays a Major Role in Mice Esophageal Hypercontractility: Reversion by Soluble Guanylyl Cyclase Stimulator.

Henrique Silva, Fabio

Impairment of Nitric Oxide Pathway by Intravascular Hemolysis Plays a Major Role in Mice Esophageal Hypercontractility: Reversion by Soluble Guanylyl Cyclase Stimulator. [electronic resource] - The Journal of pharmacology and experimental therapeutics 11 2018 - 194-202 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1521-0103

10.1124/jpet.118.249581 doi


Anemia, Sickle Cell--drug therapy
Animals
Cyclic GMP--metabolism
Esophageal Diseases--drug therapy
Esophagus--drug effects
Guanylate Cyclase--metabolism
Hemolysis--drug effects
Male
Mice
Mice, Inbred C57BL
Models, Animal
Muscle Contraction--drug effects
Muscle, Smooth--drug effects
Nitric Oxide--metabolism
Nitric Oxide Donors--pharmacology
Nitric Oxide Synthase Type III--metabolism
Nitroprusside--pharmacology
Oxidative Stress--drug effects
Phenylhydrazines--pharmacology
Pyrazoles--pharmacology
Pyridines--metabolism
Signal Transduction--drug effects
Soluble Guanylyl Cyclase--pharmacology