TGF-β signaling alters H4K20me3 status via miR-29 and contributes to cellular senescence and cardiac aging. [electronic resource]
Producer: 20190115Description: 2560 p. digitalISSN:- 2041-1723
- Aging -- genetics
- Animals
- Cellular Senescence -- genetics
- DNA Methylation -- genetics
- Embryo, Mammalian
- Epigenesis, Genetic
- Female
- Fibroblasts
- HEK293 Cells
- Heart -- physiology
- Heterocyclic Compounds, 4 or More Rings -- pharmacology
- Histone-Lysine N-Methyltransferase -- antagonists & inhibitors
- Histones -- metabolism
- Human Umbilical Vein Endothelial Cells
- Humans
- Male
- Mice
- Mice, Inbred C57BL
- MicroRNAs -- metabolism
- Myocytes, Cardiac -- physiology
- Oxidative Stress -- physiology
- Primary Cell Culture
- Signal Transduction -- genetics
- Transforming Growth Factor beta -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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