GABAergic inhibition in dual-transmission cholinergic and GABAergic striatal interneurons is abolished in Parkinson disease. [electronic resource]
Producer: 20181126Description: 1422 p. digitalISSN:- 2041-1723
- Acetylcholine -- metabolism
- Action Potentials -- drug effects
- Animals
- Bumetanide -- pharmacology
- Chlorides -- metabolism
- Cholinergic Agents -- metabolism
- Cholinergic Neurons -- drug effects
- Corpus Striatum -- drug effects
- Dopamine -- deficiency
- Gene Expression Regulation
- Glutamate Decarboxylase -- genetics
- Humans
- Inhibitory Postsynaptic Potentials -- drug effects
- Interneurons -- drug effects
- Ion Transport
- LIM-Homeodomain Proteins -- genetics
- Mice
- Mice, Transgenic
- Nerve Tissue Proteins -- genetics
- Neuronal Plasticity -- drug effects
- Parkinson Disease, Secondary -- genetics
- Patch-Clamp Techniques
- Sodium Potassium Chloride Symporter Inhibitors -- pharmacology
- Solute Carrier Family 12, Member 2 -- genetics
- Transcription Factors -- genetics
- Vesicular Inhibitory Amino Acid Transport Proteins -- genetics
- gamma-Aminobutyric Acid -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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