Disruption of a Structurally Important Extracellular Element in the Glycine Receptor Leads to Decreased Synaptic Integration and Signaling Resulting in Severe Startle Disease. [electronic resource]
Producer: 20170911Description: 7948-7961 p. digitalISSN:- 1529-2401
- Animals
- Extracellular Fluid -- metabolism
- Female
- HEK293 Cells
- Humans
- Ion Channel Gating -- physiology
- Male
- Mice
- Mice, 129 Strain
- Mice, Inbred C57BL
- Mice, Transgenic
- Motor Neurons -- metabolism
- Mutation, Missense -- physiology
- Protein Structure, Secondary
- Receptors, Glycine -- chemistry
- Severity of Illness Index
- Spinal Cord -- metabolism
- Stiff-Person Syndrome -- genetics
- Synapses -- genetics
- Synaptic Transmission -- physiology
No physical items for this record
Publication Type: Journal Article
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