Rosuvastatin postconditioning protects isolated hearts against ischemia-reperfusion injury: The role of radical oxygen species, PI3K-Akt-GSK-3β pathway, and mitochondrial permeability transition pore. [electronic resource]
Producer: 20170213Description: 3-9 p. digitalISSN:- 1755-5922
- Animals
- Antioxidants -- pharmacology
- Calcium Signaling -- drug effects
- Cytoprotection
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Glycogen Synthase Kinase 3 beta -- metabolism
- Isolated Heart Preparation
- Male
- Mitochondria, Heart -- drug effects
- Mitochondrial Membrane Transport Proteins -- antagonists & inhibitors
- Mitochondrial Permeability Transition Pore
- Myocardial Infarction -- enzymology
- Myocardial Reperfusion Injury -- enzymology
- Myocytes, Cardiac -- drug effects
- Oxidative Stress -- drug effects
- Phosphatidylinositol 3-Kinase -- metabolism
- Phosphoinositide-3 Kinase Inhibitors
- Phosphorylation
- Protein Kinase Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-akt -- metabolism
- Rats, Wistar
- Reactive Oxygen Species -- metabolism
- Rosuvastatin Calcium -- pharmacology
- Time Factors
- Ventricular Function, Left -- drug effects
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Publication Type: Journal Article
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