Rosuvastatin postconditioning protects isolated hearts against ischemia-reperfusion injury: The role of radical oxygen species, PI3K-Akt-GSK-3β pathway, and mitochondrial permeability transition pore.

Liu, Chun-Wei

Rosuvastatin postconditioning protects isolated hearts against ischemia-reperfusion injury: The role of radical oxygen species, PI3K-Akt-GSK-3β pathway, and mitochondrial permeability transition pore. [electronic resource] - Cardiovascular therapeutics Feb 2017 - 3-9 p. digital

Publication Type: Journal Article

1755-5922

10.1111/1755-5922.12225 doi


Animals
Antioxidants--pharmacology
Calcium Signaling--drug effects
Cytoprotection
Disease Models, Animal
Dose-Response Relationship, Drug
Glycogen Synthase Kinase 3 beta--metabolism
Isolated Heart Preparation
Male
Mitochondria, Heart--drug effects
Mitochondrial Membrane Transport Proteins--antagonists & inhibitors
Mitochondrial Permeability Transition Pore
Myocardial Infarction--enzymology
Myocardial Reperfusion Injury--enzymology
Myocytes, Cardiac--drug effects
Oxidative Stress--drug effects
Phosphatidylinositol 3-Kinase--metabolism
Phosphoinositide-3 Kinase Inhibitors
Phosphorylation
Protein Kinase Inhibitors--pharmacology
Proto-Oncogene Proteins c-akt--metabolism
Rats, Wistar
Reactive Oxygen Species--metabolism
Rosuvastatin Calcium--pharmacology
Time Factors
Ventricular Function, Left--drug effects