Inhibition of late sodium current attenuates ionic arrhythmia mechanism in ventricular myocytes expressing LaminA-N195K mutation. [electronic resource]
Producer: 20180103Description: 2228-2236 p. digitalISSN:- 1556-3871
- Action Potentials
- Animals
- Arrhythmias, Cardiac -- etiology
- Cardiomyopathy, Dilated -- complications
- Disease Models, Animal
- Heart Ventricles -- metabolism
- Lamin Type A -- genetics
- Mice
- Mutation, Missense
- Myocytes, Cardiac -- metabolism
- Ranolazine -- pharmacology
- Sodium Channel Blockers -- pharmacology
- Sodium Channels -- drug effects
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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