Inhibition of late sodium current attenuates ionic arrhythmia mechanism in ventricular myocytes expressing LaminA-N195K mutation.
Markandeya, Yogananda S
Inhibition of late sodium current attenuates ionic arrhythmia mechanism in ventricular myocytes expressing LaminA-N195K mutation. [electronic resource] - Heart rhythm 11 2016 - 2228-2236 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1556-3871
10.1016/j.hrthm.2016.08.007 doi
Action Potentials
Animals
Arrhythmias, Cardiac--etiology
Cardiomyopathy, Dilated--complications
Disease Models, Animal
Heart Ventricles--metabolism
Lamin Type A--genetics
Mice
Mutation, Missense
Myocytes, Cardiac--metabolism
Ranolazine--pharmacology
Sodium Channel Blockers--pharmacology
Sodium Channels--drug effects
Inhibition of late sodium current attenuates ionic arrhythmia mechanism in ventricular myocytes expressing LaminA-N195K mutation. [electronic resource] - Heart rhythm 11 2016 - 2228-2236 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1556-3871
10.1016/j.hrthm.2016.08.007 doi
Action Potentials
Animals
Arrhythmias, Cardiac--etiology
Cardiomyopathy, Dilated--complications
Disease Models, Animal
Heart Ventricles--metabolism
Lamin Type A--genetics
Mice
Mutation, Missense
Myocytes, Cardiac--metabolism
Ranolazine--pharmacology
Sodium Channel Blockers--pharmacology
Sodium Channels--drug effects