Substance P enhances tissue factor release from granulocyte-macrophage colony-stimulating factor-dependent macrophages via the p22phox/β-arrestin 2/Rho A signaling pathway. [electronic resource]
Producer: 20161025Description: 85-90 p. digitalISSN:- 1096-0961
- Acetophenones -- pharmacology
- Adipocytes -- cytology
- Amides -- pharmacology
- Aprepitant
- Arrestins -- antagonists & inhibitors
- Benzopyrans -- pharmacology
- Dynamins -- antagonists & inhibitors
- Free Radical Scavengers -- pharmacology
- Gene Expression Regulation
- Granulocyte-Macrophage Colony-Stimulating Factor -- pharmacology
- Humans
- Macrophage Activation -- drug effects
- Macrophages -- cytology
- Morpholines -- pharmacology
- NADPH Oxidases -- antagonists & inhibitors
- Neurokinin-1 Receptor Antagonists -- pharmacology
- Phosphorylcholine -- analogs & derivatives
- Primary Cell Culture
- Protein Kinase Inhibitors -- pharmacology
- Pyridines -- pharmacology
- Pyrrolidines -- pharmacology
- RNA, Small Interfering -- genetics
- Signal Transduction
- Substance P -- biosynthesis
- Thiocarbamates -- pharmacology
- Thromboplastin -- biosynthesis
- beta-Arrestin 2
- beta-Arrestins
- rho-Associated Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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