Substance P enhances tissue factor release from granulocyte-macrophage colony-stimulating factor-dependent macrophages via the p22phox/β-arrestin 2/Rho A signaling pathway.

Yamaguchi, Rui

Substance P enhances tissue factor release from granulocyte-macrophage colony-stimulating factor-dependent macrophages via the p22phox/β-arrestin 2/Rho A signaling pathway. [electronic resource] - Blood cells, molecules & diseases Mar 2016 - 85-90 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1096-0961

10.1016/j.bcmd.2016.01.006 doi


Acetophenones--pharmacology
Adipocytes--cytology
Amides--pharmacology
Aprepitant
Arrestins--antagonists & inhibitors
Benzopyrans--pharmacology
Dynamins--antagonists & inhibitors
Free Radical Scavengers--pharmacology
Gene Expression Regulation
Granulocyte-Macrophage Colony-Stimulating Factor--pharmacology
Humans
Macrophage Activation--drug effects
Macrophages--cytology
Morpholines--pharmacology
NADPH Oxidases--antagonists & inhibitors
Neurokinin-1 Receptor Antagonists--pharmacology
Phosphorylcholine--analogs & derivatives
Primary Cell Culture
Protein Kinase Inhibitors--pharmacology
Pyridines--pharmacology
Pyrrolidines--pharmacology
RNA, Small Interfering--genetics
Signal Transduction
Substance P--biosynthesis
Thiocarbamates--pharmacology
Thromboplastin--biosynthesis
beta-Arrestin 2
beta-Arrestins
rho-Associated Kinases--antagonists & inhibitors