IL-4 administration exerts preventive effects via suppression of underlying inflammation and TNF-α-induced apoptosis in steroid-induced osteonecrosis. [electronic resource]
Producer: 20170110Description: 1827-37 p. digitalISSN:- 1433-2965
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Caspase 3 -- metabolism
- Cells, Cultured
- Disease Models, Animal
- Drug Evaluation, Preclinical -- methods
- Glucocorticoids
- Inflammation -- drug therapy
- Interleukin-4 -- pharmacology
- Macrophages -- drug effects
- Methylprednisolone
- Mice, Inbred BALB C
- Osteoclasts -- drug effects
- Osteonecrosis -- chemically induced
- Recombinant Proteins -- pharmacology
- STAT1 Transcription Factor -- metabolism
- Signal Transduction -- drug effects
- Tartrate-Resistant Acid Phosphatase -- blood
- Tumor Necrosis Factor-alpha -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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