Wnt/Ryk signaling contributes to neuropathic pain by regulating sensory neuron excitability and spinal synaptic plasticity in rats. [electronic resource]
Producer: 20160913Description: 2572-2584 p. digitalISSN:- 1872-6623
- Animals
- Astrocytes -- metabolism
- Behavior, Animal -- drug effects
- Calcium Signaling
- Calcium-Calmodulin-Dependent Protein Kinase Type 2 -- drug effects
- Cyclic AMP Response Element-Binding Protein -- drug effects
- Ganglia, Spinal -- metabolism
- Hyperalgesia -- genetics
- Locomotion -- drug effects
- MAP Kinase Signaling System -- drug effects
- Male
- Nerve Fibers, Unmyelinated -- drug effects
- Neuralgia -- genetics
- Neuronal Plasticity -- drug effects
- Pain Threshold -- drug effects
- Patch-Clamp Techniques
- Posterior Horn Cells -- metabolism
- Protein Kinase C
- Rats
- Rats, Sprague-Dawley
- Real-Time Polymerase Chain Reaction
- Receptor Protein-Tyrosine Kinases -- antagonists & inhibitors
- Receptors, N-Methyl-D-Aspartate -- drug effects
- Sciatic Nerve -- injuries
- Sensory Receptor Cells -- metabolism
- Signal Transduction
- Spinal Cord -- metabolism
- Wnt Proteins -- antagonists & inhibitors
- Wnt Signaling Pathway -- drug effects
- Wnt3A Protein -- antagonists & inhibitors
- src-Family Kinases -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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