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Liu, Su

Wnt/Ryk signaling contributes to neuropathic pain by regulating sensory neuron excitability and spinal synaptic plasticity in rats. [electronic resource] - Pain Dec 2015 - 2572-2584 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

ISSN: 1872-6623

Standard No.: 10.1097/j.pain.0000000000000366 doi

Subjects--Topical Terms:
Animals
Astrocytes--metabolism
Behavior, Animal--drug effects
Calcium Signaling
Calcium-Calmodulin-Dependent Protein Kinase Type 2--drug effects
Cyclic AMP Response Element-Binding Protein--drug effects
Ganglia, Spinal--metabolism
Hyperalgesia--genetics
Locomotion--drug effects
MAP Kinase Signaling System--drug effects
Male
Nerve Fibers, Unmyelinated--drug effects
Neuralgia--genetics
Neuronal Plasticity--drug effects
Pain Threshold--drug effects
Patch-Clamp Techniques
Posterior Horn Cells--metabolism
Protein Kinase C
Rats
Rats, Sprague-Dawley
Real-Time Polymerase Chain Reaction
Receptor Protein-Tyrosine Kinases--antagonists & inhibitors
Receptors, N-Methyl-D-Aspartate--drug effects
Sciatic Nerve--injuries
Sensory Receptor Cells--metabolism
Signal Transduction
Spinal Cord--metabolism
Wnt Proteins--antagonists & inhibitors
Wnt Signaling Pathway--drug effects
Wnt3A Protein--antagonists & inhibitors
src-Family Kinases--drug effects