ST2 receptor invalidation maintains wound inflammation, delays healing and increases fibrosis. [electronic resource]
Producer: 20170110Description: 71-4 p. digitalISSN:- 1600-0625
- Animals
- Cell Differentiation
- Cell Nucleus -- metabolism
- Fibrosis -- metabolism
- Flow Cytometry
- Inflammation -- physiopathology
- Interleukin-1 Receptor-Like 1 Protein -- metabolism
- Interleukin-33 -- metabolism
- Macrophages -- metabolism
- Mice
- Mice, Inbred BALB C
- Neovascularization, Pathologic
- Neutrophils -- metabolism
- Protein Isoforms
- RNA -- metabolism
- Wound Healing
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Publication Type: Letter; Research Support, Non-U.S. Gov't
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