ST2 receptor invalidation maintains wound inflammation, delays healing and increases fibrosis.
Lee, James S
ST2 receptor invalidation maintains wound inflammation, delays healing and increases fibrosis. [electronic resource] - Experimental dermatology Jan 2016 - 71-4 p. digital
Publication Type: Letter; Research Support, Non-U.S. Gov't
1600-0625
10.1111/exd.12833 doi
Animals
Cell Differentiation
Cell Nucleus--metabolism
Fibrosis--metabolism
Flow Cytometry
Inflammation--physiopathology
Interleukin-1 Receptor-Like 1 Protein--metabolism
Interleukin-33--metabolism
Macrophages--metabolism
Mice
Mice, Inbred BALB C
Neovascularization, Pathologic
Neutrophils--metabolism
Protein Isoforms
RNA--metabolism
Wound Healing
ST2 receptor invalidation maintains wound inflammation, delays healing and increases fibrosis. [electronic resource] - Experimental dermatology Jan 2016 - 71-4 p. digital
Publication Type: Letter; Research Support, Non-U.S. Gov't
1600-0625
10.1111/exd.12833 doi
Animals
Cell Differentiation
Cell Nucleus--metabolism
Fibrosis--metabolism
Flow Cytometry
Inflammation--physiopathology
Interleukin-1 Receptor-Like 1 Protein--metabolism
Interleukin-33--metabolism
Macrophages--metabolism
Mice
Mice, Inbred BALB C
Neovascularization, Pathologic
Neutrophils--metabolism
Protein Isoforms
RNA--metabolism
Wound Healing