Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression. [electronic resource]

By: Contributor(s): Producer: 20151102Description: 870-5 p. digitalISSN:
  • 1090-2104
Subject(s): Online resources: In: Biochemical and biophysical research communications vol. 463
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't

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