Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression.
Kikuchi, Hidehiko
Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression. [electronic resource] - Biochemical and biophysical research communications Aug 2015 - 870-5 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1090-2104
10.1016/j.bbrc.2015.06.027 doi
Animals
Apoptosis--drug effects
Cell Line
Chickens
Endoplasmic Reticulum--drug effects
Genes, bcl-2
Histone Acetyltransferases--genetics
Thapsigargin--pharmacology
Up-Regulation
Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression. [electronic resource] - Biochemical and biophysical research communications Aug 2015 - 870-5 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1090-2104
10.1016/j.bbrc.2015.06.027 doi
Animals
Apoptosis--drug effects
Cell Line
Chickens
Endoplasmic Reticulum--drug effects
Genes, bcl-2
Histone Acetyltransferases--genetics
Thapsigargin--pharmacology
Up-Regulation