Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression.

Kikuchi, Hidehiko

Lack of GCN5 remarkably enhances the resistance against prolonged endoplasmic reticulum stress-induced apoptosis through up-regulation of Bcl-2 gene expression. [electronic resource] - Biochemical and biophysical research communications Aug 2015 - 870-5 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1090-2104

10.1016/j.bbrc.2015.06.027 doi


Animals
Apoptosis--drug effects
Cell Line
Chickens
Endoplasmic Reticulum--drug effects
Genes, bcl-2
Histone Acetyltransferases--genetics
Thapsigargin--pharmacology
Up-Regulation