Truncation and activation of GSK-3β by calpain I: a molecular mechanism links to tau hyperphosphorylation in Alzheimer's disease. [electronic resource]
Producer: 20151201Description: 8187 p. digitalISSN:- 2045-2322
- Alzheimer Disease -- metabolism
- Animals
- Brain -- drug effects
- Calcium -- metabolism
- Calpain -- metabolism
- Glycogen Synthase Kinase 3 -- genetics
- Glycogen Synthase Kinase 3 beta
- HEK293 Cells
- Humans
- Kainic Acid -- toxicity
- Male
- Mice
- Phosphorylation -- drug effects
- Proteolysis
- Recombinant Fusion Proteins -- genetics
- Up-Regulation -- drug effects
- tau Proteins -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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