Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance. [electronic resource]
Producer: 20150512Description: H637-50 p. digitalISSN:- 1522-1539
- Action Potentials
- Animals
- Calcium -- metabolism
- Calcium Signaling
- Cardiomyopathies -- chemically induced
- Disease Models, Animal
- Doxorubicin
- Energy Metabolism
- HEK293 Cells
- Humans
- Mice, Inbred C57BL
- Mice, Knockout
- Mitochondria, Heart -- metabolism
- Mutation
- Myocardial Contraction
- Myocardial Reperfusion Injury -- genetics
- Myocytes, Cardiac -- metabolism
- Oxidative Stress
- Oxygen Consumption
- Reactive Oxygen Species -- metabolism
- TRPM Cation Channels -- deficiency
- Time Factors
- Transfection
- Ventricular Function, Left
- Ventricular Pressure
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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