Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.
Hoffman, Nicholas E
Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance. [electronic resource] - American journal of physiology. Heart and circulatory physiology Mar 2015 - H637-50 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1522-1539
10.1152/ajpheart.00720.2014 doi
Action Potentials
Animals
Calcium--metabolism
Calcium Signaling
Cardiomyopathies--chemically induced
Disease Models, Animal
Doxorubicin
Energy Metabolism
HEK293 Cells
Humans
Mice, Inbred C57BL
Mice, Knockout
Mitochondria, Heart--metabolism
Mutation
Myocardial Contraction
Myocardial Reperfusion Injury--genetics
Myocytes, Cardiac--metabolism
Oxidative Stress
Oxygen Consumption
Reactive Oxygen Species--metabolism
TRPM Cation Channels--deficiency
Time Factors
Transfection
Ventricular Function, Left
Ventricular Pressure
Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance. [electronic resource] - American journal of physiology. Heart and circulatory physiology Mar 2015 - H637-50 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1522-1539
10.1152/ajpheart.00720.2014 doi
Action Potentials
Animals
Calcium--metabolism
Calcium Signaling
Cardiomyopathies--chemically induced
Disease Models, Animal
Doxorubicin
Energy Metabolism
HEK293 Cells
Humans
Mice, Inbred C57BL
Mice, Knockout
Mitochondria, Heart--metabolism
Mutation
Myocardial Contraction
Myocardial Reperfusion Injury--genetics
Myocytes, Cardiac--metabolism
Oxidative Stress
Oxygen Consumption
Reactive Oxygen Species--metabolism
TRPM Cation Channels--deficiency
Time Factors
Transfection
Ventricular Function, Left
Ventricular Pressure