Increasing cisplatin sensitivity by schedule-dependent inhibition of AKT and Chk1. [electronic resource]
Producer: 20150916Description: 1600-12 p. digitalISSN:- 1555-8576
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Cell Cycle Checkpoints -- drug effects
- Cell Line, Tumor
- Checkpoint Kinase 1
- Cisplatin -- pharmacology
- Cyclin-Dependent Kinase Inhibitor p27 -- genetics
- DNA Damage -- drug effects
- Drug Resistance, Neoplasm
- Enzyme Activation
- Gene Expression Regulation, Neoplastic
- Gene Knockdown Techniques
- Heterocyclic Compounds, 3-Ring -- pharmacology
- Humans
- Phosphorylation
- Protein Kinase Inhibitors -- pharmacology
- Protein Kinases -- metabolism
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- RNA Interference
- RNA, Small Interfering -- genetics
- Tumor Suppressor Protein p53 -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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