Inflammation and hyperglycemia mediate Deaf1 splicing in the pancreatic lymph nodes via distinct pathways during type 1 diabetes. [electronic resource]
Producer: 20150331Description: 604-17 p. digitalISSN:- 1939-327X
- Aging
- Alternative Splicing
- Animals
- Blood Glucose
- Cell Cycle Proteins -- genetics
- DNA-Binding Proteins
- Diabetes Mellitus, Type 1 -- genetics
- Female
- HEK293 Cells
- Humans
- Hyperglycemia -- metabolism
- Inflammation -- metabolism
- Islets of Langerhans -- cytology
- Lymph Nodes -- physiology
- Mice
- Mice, Inbred NOD
- Mice, SCID
- Nerve Tissue Proteins -- genetics
- Nuclear Proteins -- genetics
- Pancreas -- physiology
- Pancreatic Elastase -- genetics
- Polypyrimidine Tract-Binding Protein -- genetics
- RNA-Binding Proteins -- genetics
- Repressor Proteins -- genetics
- Serine-Arginine Splicing Factors
- Spleen -- cytology
- Transcription Factors -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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