Halofuginone-induced amino acid starvation regulates Stat3-dependent Th17 effector function and reduces established autoimmune inflammation. [electronic resource]
Producer: 20140418Description: 2167-76 p. digitalISSN:- 1550-6606
- Amino Acids -- deficiency
- Animals
- Encephalomyelitis, Autoimmune, Experimental -- genetics
- Inflammation -- chemically induced
- Interleukin-23 -- genetics
- Lymphocyte Activation
- Mice
- Mice, Inbred BALB C
- Mice, Knockout
- Phosphorylation -- genetics
- Piperidines
- Quinazolinones
- Receptors, CCR6 -- genetics
- Receptors, Interleukin -- genetics
- STAT3 Transcription Factor -- genetics
- Th17 Cells -- immunology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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