Tumor necrosis factor-α accelerates the resolution of established pulmonary fibrosis in mice by targeting profibrotic lung macrophages. [electronic resource]
Producer: 20140617Description: 825-37 p. digitalISSN:- 1535-4989
- Animals
- Bleomycin
- Cells, Cultured
- Disease Models, Animal
- Hydroxyproline -- metabolism
- Idiopathic Pulmonary Fibrosis -- drug therapy
- Lung -- drug effects
- Lung Compliance
- Macrophages, Alveolar -- drug effects
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Mice, Transgenic
- Receptor, Macrophage Colony-Stimulating Factor -- genetics
- Recovery of Function
- Remission Induction
- Tacrolimus Binding Proteins -- genetics
- Time Factors
- Tumor Necrosis Factor-alpha -- deficiency
- fas Receptor -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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