PPAR-γ activation by rosiglitazone suppresses angiotensin II-mediated proliferation and phenotypictransition in cardiac fibroblasts via inhibition of activation of activator protein 1. [electronic resource]
Producer: 20140414Description: 196-203 p. digitalISSN:- 1879-0712
- Angiotensin II -- pharmacology
- Animals
- Cell Differentiation -- drug effects
- Cell Proliferation -- drug effects
- Cell Survival -- drug effects
- Collagen Type I -- genetics
- Collagen Type III -- genetics
- Cyclin D1 -- genetics
- Fibroblasts -- cytology
- JNK Mitogen-Activated Protein Kinases -- chemistry
- Myocardium -- cytology
- Myofibroblasts -- cytology
- PPAR gamma -- metabolism
- Phenotype
- Protein Multimerization -- drug effects
- Protein Structure, Quaternary
- Proto-Oncogene Proteins c-fos -- chemistry
- RNA, Messenger -- genetics
- Rats
- Rats, Wistar
- Rosiglitazone
- Thiazolidinediones -- pharmacology
- Transcription Factor AP-1 -- metabolism
- Transforming Growth Factor beta1 -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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