HDAC turnover, CtIP acetylation and dysregulated DNA damage signaling in colon cancer cells treated with sulforaphane and related dietary isothiocyanates. [electronic resource]
Producer: 20140106Description: 612-23 p. digitalISSN:- 1559-2308
- Acetylation
- Antineoplastic Agents -- pharmacology
- Apoptosis
- Autophagy
- Carrier Proteins -- metabolism
- Cell Cycle Checkpoints
- Cell Line
- Cell Line, Tumor
- Colon -- cytology
- Colonic Neoplasms -- genetics
- DNA Damage
- Endodeoxyribonucleases
- Gene Expression
- Histone Deacetylase Inhibitors -- pharmacology
- Histone Deacetylases -- genetics
- Humans
- Isothiocyanates -- administration & dosage
- Nuclear Proteins -- metabolism
- Sulfoxides
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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