HDAC turnover, CtIP acetylation and dysregulated DNA damage signaling in colon cancer cells treated with sulforaphane and related dietary isothiocyanates.

Rajendran, Praveen

HDAC turnover, CtIP acetylation and dysregulated DNA damage signaling in colon cancer cells treated with sulforaphane and related dietary isothiocyanates. [electronic resource] - Epigenetics Jun 2013 - 612-23 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural

1559-2308

10.4161/epi.24710 doi


Acetylation
Antineoplastic Agents--pharmacology
Apoptosis
Autophagy
Carrier Proteins--metabolism
Cell Cycle Checkpoints
Cell Line
Cell Line, Tumor
Colon--cytology
Colonic Neoplasms--genetics
DNA Damage
Endodeoxyribonucleases
Gene Expression
Histone Deacetylase Inhibitors--pharmacology
Histone Deacetylases--genetics
Humans
Isothiocyanates--administration & dosage
Nuclear Proteins--metabolism
Sulfoxides