Reelin, an extracellular matrix protein linked to early onset psychiatric diseases, drives postnatal development of the prefrontal cortex via GluN2B-NMDARs and the mTOR pathway. [electronic resource]
Producer: 20141117Description: 417-26 p. digitalISSN:- 1476-5578
- Analysis of Variance
- Animals
- Animals, Newborn
- Cell Adhesion Molecules, Neuronal -- deficiency
- Conditioning, Classical -- drug effects
- Dendritic Spines -- drug effects
- Dose-Response Relationship, Drug
- Excitatory Amino Acid Agents -- pharmacology
- Extinction, Psychological -- drug effects
- Extracellular Matrix Proteins -- deficiency
- Fear -- drug effects
- Female
- In Vitro Techniques
- Ketamine -- pharmacology
- Long-Term Potentiation -- drug effects
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Nerve Tissue Proteins -- deficiency
- Patch-Clamp Techniques
- Phenols
- Piperidines -- pharmacology
- Prefrontal Cortex -- cytology
- Receptors, N-Methyl-D-Aspartate -- metabolism
- Reelin Protein
- Serine Endopeptidases -- deficiency
- Signal Transduction -- drug effects
- Sirolimus
- TOR Serine-Threonine Kinases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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