Gain of interaction with IRS1 by p110α-helical domain mutants is crucial for their oncogenic functions. [electronic resource]
Producer: 20130722Description: 583-93 p. digitalISSN:- 1878-3686
- Amino Acid Substitution
- Cell Line, Tumor
- Class I Phosphatidylinositol 3-Kinases
- Humans
- Insulin Receptor Substrate Proteins -- chemistry
- Neoplasms -- genetics
- Phosphatidylinositol 3-Kinases -- chemistry
- Phosphorylation
- Protein Interaction Mapping
- Protein Structure, Tertiary
- Proto-Oncogene Proteins c-akt -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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