Cathepsin B and uPAR regulate self-renewal of glioma-initiating cells through GLI-regulated Sox2 and Bmi1 expression. [electronic resource]
Producer: 20130419Description: 550-9 p. digitalISSN:- 1460-2180
- AC133 Antigen
- Animals
- Antigens, CD -- metabolism
- Cathepsin B -- genetics
- Cell Line, Tumor
- Cell Proliferation
- Cell Separation
- Female
- Flow Cytometry
- Gene Expression
- Gene Expression Regulation, Neoplastic
- Gene Knockdown Techniques
- Glioma -- metabolism
- Glycoproteins -- metabolism
- Hedgehog Proteins -- metabolism
- Humans
- Mice
- Mice, Nude
- Neoplasm Transplantation
- Neoplastic Stem Cells -- metabolism
- Peptides -- metabolism
- Polycomb Repressive Complex 1 -- genetics
- RNA, Small Interfering -- genetics
- Receptors, Urokinase Plasminogen Activator -- genetics
- SOXB1 Transcription Factors -- genetics
- Signal Transduction
- Transcription Factors -- metabolism
- Transcriptional Activation
- Zinc Finger Protein GLI1
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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