Selective binding of nuclear alpha-synuclein to the PGC1alpha promoter under conditions of oxidative stress may contribute to losses in mitochondrial function: implications for Parkinson's disease. [electronic resource]
Producer: 20121221Description: 993-1003 p. digitalISSN:- 1873-4596
- Aged
- Animals
- Cell Nucleus -- metabolism
- Chromatin -- metabolism
- Chromatin Immunoprecipitation
- Electron Transport Complex I -- metabolism
- Heat-Shock Proteins -- genetics
- Humans
- Mice
- Mice, Transgenic
- Middle Aged
- Mitochondria -- metabolism
- Oxidative Stress
- PC12 Cells
- Parkinson Disease -- metabolism
- Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
- Promoter Regions, Genetic
- Protein Binding
- Rats
- Transcription Factors -- genetics
- alpha-Synuclein -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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