Complement C3a-induced IL-17 plays a critical role in an IgE-mediated late-phase asthmatic response and airway hyperresponsiveness via neutrophilic inflammation in mice. [electronic resource]
Producer: 20120814Description: 5694-705 p. digitalISSN:- 1550-6606
- Animals
- Antibodies, Monoclonal -- physiology
- Asthma -- immunology
- Bronchial Hyperreactivity -- immunology
- CD4-Positive T-Lymphocytes -- immunology
- Complement C3a -- antagonists & inhibitors
- Immunoglobulin E -- physiology
- Inflammation Mediators -- antagonists & inhibitors
- Interleukin-17 -- biosynthesis
- Male
- Mice
- Mice, Inbred BALB C
- Neutrophils -- immunology
- Receptors, Complement -- antagonists & inhibitors
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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