A UVR-induced G2-phase checkpoint response to ssDNA gaps produced by replication fork bypass of unrepaired lesions is defective in melanoma. [electronic resource]
Producer: 20120717Description: 1681-8 p. digitalISSN:- 1523-1747
- CDC2 Protein Kinase -- metabolism
- Cell Cycle Checkpoints -- genetics
- Cell Line, Tumor
- Cyclin B1 -- metabolism
- DNA Replication -- genetics
- DNA, Single-Stranded -- genetics
- DNA-Binding Proteins -- physiology
- Epidermal Cells
- Epidermis -- radiation effects
- G1 Phase -- genetics
- G2 Phase -- genetics
- Humans
- Melanoma -- genetics
- Mutation -- genetics
- S Phase -- genetics
- Skin Neoplasms -- genetics
- Ultraviolet Rays -- adverse effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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