Depletion of RUNX1/ETO in t(8;21) AML cells leads to genome-wide changes in chromatin structure and transcription factor binding. [electronic resource]
Producer: 20121119Description: 1829-41 p. digitalISSN:- 1476-5551
- Acetylation
- Binding Sites
- CCAAT-Binding Factor -- genetics
- Cell Differentiation -- genetics
- Cell Line, Tumor
- Chromatin -- genetics
- Chromosomes, Human, Pair 21
- Chromosomes, Human, Pair 8
- Cluster Analysis
- Core Binding Factor Alpha 2 Subunit -- genetics
- Gene Expression Profiling
- Gene Silencing
- Histones -- metabolism
- Humans
- Leukemia, Myeloid, Acute -- genetics
- Mutation
- Oncogene Proteins, Fusion -- genetics
- Protein Binding
- Proto-Oncogene Proteins -- genetics
- RNA Polymerase II -- metabolism
- RUNX1 Translocation Partner 1 Protein
- Transcription Factors -- genetics
- Transcriptional Activation
- Translocation, Genetic
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.