Depletion of RUNX1/ETO in t(8;21) AML cells leads to genome-wide changes in chromatin structure and transcription factor binding.

Ptasinska, A

Depletion of RUNX1/ETO in t(8;21) AML cells leads to genome-wide changes in chromatin structure and transcription factor binding. [electronic resource] - Leukemia Aug 2012 - 1829-41 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

1476-5551

10.1038/leu.2012.49 doi


Acetylation
Binding Sites
CCAAT-Binding Factor--genetics
Cell Differentiation--genetics
Cell Line, Tumor
Chromatin--genetics
Chromosomes, Human, Pair 21
Chromosomes, Human, Pair 8
Cluster Analysis
Core Binding Factor Alpha 2 Subunit--genetics
Gene Expression Profiling
Gene Silencing
Histones--metabolism
Humans
Leukemia, Myeloid, Acute--genetics
Mutation
Oncogene Proteins, Fusion--genetics
Protein Binding
Proto-Oncogene Proteins--genetics
RNA Polymerase II--metabolism
RUNX1 Translocation Partner 1 Protein
Transcription Factors--genetics
Transcriptional Activation
Translocation, Genetic